Neena Khanna Dermatology Pdf 118
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This book is focused on subjects such as leprosy, pigmentary problems, mycological diseases and sexually transmitted diseases in the field of immunology of leprosy. Some groupings have been made morphologically to emphasize how dermatoses, with different etiogenesis, may simulate each other. The physician is constantly faced with the challenge of diagnosing and treating exotic conditions. The projection in this atlas of skin lesions on a pigmented background will aid the physician in his diagnosis. The book is divided into several chapters with various diseases conventionally grouped some unconventional grouping has also been made to emphasize on close clinical simulation. A chapter on sexually transmitted diseases has been included since, in many countries, the specialties of dermatology and venereology. This book contains twenty five chapter, and each chapter gives brief information of diseases such as pyodermas, Fungal Infections, Viral Infections, Leprosy, cutaneous tuberculosis, diseases caused by Arthropods and Parasites. Moreover, description of diseases like dermatitis and Eczema, Abnormal Vascular Responses, Abnormal Vascular Responses, Autoimmune vesiculobullous dermatoses, metabolic and nutritional dermatoses, Disorders of Skin Pigmentation, the Connective tissue diseases, disorders due to Physical Agents, Diseases of the Skin Appendages are also included in the book.
X-linked chronic granulomatous disease (CGD) is caused by mutations in the CYBB gene, which encodes for gp91phox, a protein in the NADPH oxidase complex. Patients with pathogenic missense variants that encode amino acids 1-309 for gp91phox are associated with residual superoxide production and reduced risk of infection, compared to more severely affected missense mutations involving amino acids 310-570 with nearly absent superoxide production. We report a 3-year-old male who initially presented to dermatology with a history of severe diaper dermatitis that appeared at 12 months of age and gradually progressed to a diffuse, erythematous, non-pruritic dermatitis accompanied by alopecia universalis by 18 months. Parents reported that he did not perspire, and his skin would become warm and red with activity, suggestive of hypohidrosis. Patient failed topical and oral antifungals therapy. Topical steroids provided some improvement, with recurrence of rash after therapy discontinuation. Due to occipital and axillary lymphadenopathy, a lymph node biopsy was obtained, which demonstrated increased interfollicular histiocytes. Skin biopsy of the scalp demonstrated inflammation around deep hair follicles and eccrine sweat glands, composed of abundant neutrophils and smaller numbers of lymphocytes and histiocytes, with neutrophilic inflammation more sparsely involving the superficial dermis and epidermis. Both biopsies were culture negative. Clinical exome sequencing identified a novel hemizygous missense variant in the CYBB gene (c.901G>T; pVal301Phe). A dihydrorhodamine (DHR) assay was performed which demonstrated reduced NADPH oxidase activity with patient stimulation index (SI) of 14.7, control SI = 124.4. He has no infectious history, likely due to presence of residual oxidative burst activity. The lymphadenopathy and diffuse dermatitis resolved with a prolonged steroid taper over 9 months. He has started to perspire with activities and has had some sparse hair growth. Due to his prolonged, severe inflammatory dermatitis, hematopoietic stem cell transplant is planned utilizing a matched sibling donor for definitive cure. 2b1af7f3a8